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Multiple endocrine neoplasia type 2 Medullary thyroid cancer RET variants Stage II colorectal cancer Develop disease relapse Rectal carcinoma Psoas muscle volume Geriatric nutritional risk index SY6GAL1 gene Unfavorable prognosis Astrocytoma risk Seizure diary Initially aggravate symptoms Plasmin-α2 plasmin inhibitor complex Symptoms including seizures Soluble fibrin Immunotherapy resistance Survival-related mutations Thrombin-antithrombin III complex Supine immobilization devices Apoptotic pathways Skin dose Essential factors Immunotherapeutic efficacies Gastric tumor bleeding Imaging biomarkers Neoplasm invasion Nomogram model Tumor thickness Prolong survival Radio-genomics Adjuvant surgery Concurrent radiotherapy Cell type Substantial increase Cancer critical Traumatic surgical intensive Prophylactic management Genomic sequencing classifier EGFR family Human tumorigenesis Beneficial biomarkers Tyrosine kinase I subfamily Pulmonary metastasectomy Institutional experience Metastatic sarcomas Carnitine palmitoyl transferase 1A Higher-order genetic interaction Marginal effect Plus enzalutamide Multiple glycolysisrelated genes MiRNA-mRNA pairs Colorectal peritoneal metastases Surgical staging Selecting patients AFP-negative HCC patients Nude rat xenograft tumor model IgG4 antibody therapy ROS-stimulating therapy TACSTD2 hypermethylation Main function Glia maturation factor-γ Trastuzumab administration Bibliographic database HER2-positive breast tumors Construct competing endogenous RNA Axillary non-SLN metastases Serum tumor markers Endoscopic technology Dual probe difference specimen imaging Inhibit EMT process BMP pathway Immunological effect Various cancer types Xenografted tumor development Glottic cancer Vocal cord Increase survival rate Desmoid tumor fibromatosis Conservative treatments Migration capacities Colonogenic assays Therapeutic guide Cancer-special survival Prognostic efficacy Establish nomogram models Actual observations Line of therapy Age-standardized mortality rate Breast cancer-specific survival Still controversial RAS family protooncogenes Including KRAS Molecular abnormalities Real-world study Uveal nevus Activating Gαq signalling mutations Tumour inflammation Chemotherapy alone Special attention

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business or provide - 4 sao (17 lượt)